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KRAS -mutant cancers exhibit a pronounced dependence on antioxidant adaptations [39, 40], and evasion or inactivation of ferroptosis contributes to Kras-driven tumor development and progression [41, 42]

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This growth hormone releasing hormone analog works by binding to GHRH receptors in the anterior pituitary, triggering pulsatile GH secretion that mimics the body's natural rhythms